tox, It's a good point, but target plant selectivity is a hallmark of herbicide development. What everyone wants is high effectiveness with only the target plant, and no effect on the crop being protected. For clopyralid, as an example, forage and grass hays are permitted a residual level of 500 ppm. We know that tomatoes are sensitive to levels of 30 ppb in the soil. If you couple that kind of effective selectivity with some environemtal persistence, then the stage gets set for someone, somewhere to have an issue. edit - To illustrate the point -- if the half life for degradation were 30 days (about the average for clopyralid), and the soil level were 10 ppm, then by my calculation it would take ~9 months for that 10 ppm to decay to a level of While farmers did use to love the persistent aspect, many are now questioning the desirability because of how their options can get cut when faced with a need for crop rotation. That hit ND wheat farmers when scab became an issue. I have heard anecdotally that WI dairy farmers in some areas are now prohibited from exporting their manures off-farm, and many are running out of places to keep the stuff. My suspicion is that market forces in the ag world may set a new direction before EPA and registration become a major issue. This post was edited by TXEB on Thu, May 2, 13 at 12:44

This plant belongs to Capsicum family.In fact I harvested plenty of peppers from the same plant during last summer.As I have mentioned earlier that this plant was transplanted from back yard garden at the end of summer of 2007 before the on set of freezing temperature to raise as pereniel plant.Plant is in sun room and about 9 months old producing bunch of flowers.I intend to take the plant out door by end of May 2008 by that time plant will be one year old.Since there are plenty of flowers and I am not sure the flowers will produce any fruits at this time I thought to use the flowers in salad.Few of the flowers have already droped.

Hello Battalina - A couple of thoughts regarding your posting... You mentioned that : "Some of them had dried up bluberry looking black fruit on them." Poison Ivy (Toxicodendron radicans) does NOT have dried up looking blueberry type fruits , although Parthenocissus quinquefolia (commonly known as Virginia creeper and Parthenocissus tricuspida aka Boston Ivy)do. There are some people who have an allergic type reaction from Parthenoccissus species although in my case , although I am extremely allergic to Toxicodendron species , I handle Partheocissus species frequently with no bad reaction at all. The plants I mentioned are frequently encountered growing together. The offending plants are best removed during the Winter (when urushiol levels will be lowest) by an individual who is not allergic by pulling / digging the roots out of the ground. The oil in the Poison Ivy is what some people are allergic to and the stems during the winter don't have as much sap in them as they do during the summer. The oil (aka catechol) is released most easily from bruised leaves or any other part which will place tiny droplets of the oil into the air (where it may land on the skin, clothes or be breathed into the respiratory system. People who are extremely allergic to Toxicodendron species can get the allergic dermatitis simply from being near the plants as a certain amount of the oil is evaporating into the atmosphere , most especially on hot summer days. People who are extremely allergic to Toxicodendron can pick up the oil from the fur of pets that have brushed up against the leaves , from clothing or anything (!) that has the oil on it and can transfer it to the allergic person. People who are extremely allergic to Toxicodendron catechol can get it from other people while the exudate from the encrusted ares is still yellow and it can definitely be spread to other people or other parts of your body from the oil being transferred from any contaminated object e.g., q-tips, washcloths, bedsheets, etc. The Poison Ivy Catechol is the aromatic portion of some of our neurotransmitters and the formula in brief is catechol + an amine = catecholamine (!)...The human catecholamines are serotonin, dopamine, epinephine, norepinephrine, metanephrine and normetanephrine. The catechol oil in different species of Toxicodendron and plants with related catechols consists of a mixture of different oils, so that is why some people are allergic to Poison Ivy but not Poison Sumac because the ratio of the different oil fractionations is different. Poison Ivy dermatitis may one day act as an indicator of certain types of auto-immune disorders. I am fully aware that some of what I stated is listed as untrue by some dermatological sources although what I have stated regarding extremely allergic people is based upon my own hyper-allergic experience and research regarding my own condition. I have found that most practicing dermatologists will not know about the specific info I have shared, although a few doing specialized / focused research into urushiol related toxicity / allergicity might... I have added a link to a similar post on plants in the Sumac Family including Toxicodendron and edibles like cashews , pistachios and mangos. I hope my offerings may prove to be helpful... Here is a link that might be useful: Poison Ivy (Toxicodendron radicans) identification dermatitis rash prevention and remedial cures info

I also have not received any seeds. I've seen no effort on Olivia's part to contact me. For what it is worth, I have participated in quite a few swaps within this community before and have always found them worth taking the chance...sometimes fantastically so. However, I walked into the swaps recognizing that it IS taking a chance, especially if someone is a first-timer at facilitating a swap, as in this case apparently. I don't plan to stop participating in swaps in the future, but find it wise to gamble no more than I can afford to lose.